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Moult PJA, Rees LH, Besser GM

Pathophysiology of inflammation in cerebral X-ALD New and old concepts suggest that the inability to efficiently degrade VLCFA, as well as the incorporation of VLCFA into different complex lipids, is directly involved in the pathology of the cerebral inflammatory form of X-ALD

10.1016/j.bbalip.2018.07.007

doi: 10.1093/rb/rbad108 287 MohantyCSahooSK

To this end, using a perifusion system adapted to NMR spectrometer (Roby et al., 1987), we first followed the effects of in vivo transition from normoxia to hypoxia, and vice versa , on the energy metabolism of 4 wpi nodules

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