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In prostate cancer, androgen deprivation triggers AR dissociation from the CEBPB promoter, enabling histone acetylation and active transcription of CEBPB, fostering castration resistance [73]
Fluorescence and protease sensitivity assays show that oleoylethanolamide, a PPAR agonist (Fu et al., 2003), serves as a direct ligand for NHR-80, but not NHR-49 (Folick et al., 2015)
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16th EORTC-NCI-AACR Symposium on Molecular Targets and Cancer Therapeutics
