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Journal of Hepatology 34 (2001) 946-954

In its initial compensatory phase, the UPR alleviates ER stress by suppressing global protein synthesis, upregulating chaperone levels, and facilitating the degradation of misfolded proteins

Moreover, GSH increase has been positively correlated with HepG2 cell growth: inhibiting or increasing cellular GSH, DNA synthesis and cell proliferation were, respectively, reduced or improved [319]

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Feghali-Bostwick CA, Gadgil AS, Otterbein LE, et al

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