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This review integrates current evidence on how brain peptides contribute to AD pathophysiology, summarizes recent progress in peptide-based therapeutic strategies and delivery platforms, and critically examines the remaining barriers to clinical translation, including bloodbrain barrier penetration, metabolic stability, off-target effects, and the need for biomarker-guided patient stratification

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Treatments include foliar application of lead at two levels (0.0 and 1 mmol) as lead acetate, foliar application of liquid sulfur at two levels (0.0 and 2 per thousand), and foliar application of amino acids at four levels (0.0, serine at 200 mg/L, glutamine at 200 mg/L, and co-application of serine and glutamine at the same concentrations) at pre-flowering stage

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Conclusions Our findings demonstrate that screening potential natural products through an AD-related pathway-gene network is a promising strategy for discovering novel therapeutics for AD