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These insights from CNS pathophysiology illuminate that disruption of iron levels, by removing their buffering capacity via targeting of ferritin, could provide a beneficial role in sensitizing brain cancers to iron-dependent cell death and reduced proliferation, as we will explore in this review

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Clin Exp Nephrol 9:127131 Elkanzi NAA, Hrichi H, Alolayan RA, Derafa W, Zahou FM, Bakr RB (2022) Synthesis of chalcones derivatives and their biological activities: a review

Delivery of siRNA to the mouse brain by systemic injection of targeted exosomes