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ROS/RNS contributes to rearranging macrophage differentiation and exerting their effector functions in response to tissue environments via intermediating the main cellular oxidation-reduction (redox) pathways, including glutathione (GSH) pathways, and NF-E2-related factor 2 (NRF2) [30, 63, 64]

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Effect of iron chelation on myocardial infarct size and oxidative stress in ST-elevation-myocardial infarction