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SPMs act as receptor agonists at specific Gprotein coupled receptors (e.g., ALX/FPR2 for lipoxin A 4 and RvD1, ChemR23/CMKLR1 for RvE1, and GPR32 for RvD1) to reprogram leukocyte responses and reduce proinflammatory cytokine production while enhancing clearance of apoptotic cells and microbial debris, a mechanism that differs fundamentally from cytokine blockade or antimicrobial killing [114, 115, 116]

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When combining peptides, starting with lower individual doses and gradually adjusting based on response is advisable

Superoxide dismutase and glutathione peroxidase were reduced, while the activities of catalase, glutathione reductase, and myeloperoxidase were found to be elevated [275]

Li et al., 2022)

Int J Vitam Nutr Res 81(4):197203