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Acknowledgments Funding for this work was provided by TandemRain Innovations (Vancouver, WA)

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Exogenous induction of NO treatment demonstrates an inhibitory role against NEPC functions Next, we studied how NO treatment could disrupt the functional properties of high-grade PCa cells

In chronic kidney disease, loss of kidney epithelial cells leads to glomerulosclerosis and tubular atrophy, whereas interstitial inflammation and fibrosis result from an excess of leukocytes and myofibroblasts
