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Description
Our drug-gene interaction analysis identified several promising therapeutic compounds, with erastin showing particularly strong binding affinity to SLC7A11
However, C16-carnitine disrupts this interaction, resulting in inefficient uptake of Ca 2+ released from the SR by mitochondria, thereby exacerbating intracellular calcium overload [31]
New strategy for MS treatment with autoantigen-modified liposomes and their therapeutic effect
Mechanisms of Action : GHK-Cu works by stimulating regenerative processes
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(PubMed) Chiang EC, Shen S, Kengeri SS, et al
