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In future trials it will be important to keep track of comorbidities for clinical trial design, either for the purpose of post hoc subgroup analysis or a priori subgroup recruitment, since the neuroathophysiological mechanisms in IC/ BPS patients with different co-morbidities are likely to be different

The immunopathogenesis is driven by a predominance of T helper (Th)1-mediated immune responses, with elevated interferon- (IFN-) production and impaired function of regulatory T cells (Tregs), which normally restrain autoreactive lymphocytes [11,12]

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Who should not undergo a bone marrow transplant

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A slower dose increase or temporary dose adjustment may help improve tolerance