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ZNT treatment reduced HIF-1 expression levels to 2.5-fold, suggesting a mitigating effect on hypoxia-induced transcriptional activity

Several pathogens capable of multiplying within the phagosomal compartment of PMNs or other phagocytes secrete proteins or toxins that prevent the activated NADPH oxidase complex from assembling at the phagosomal membrane, effectively blocking oxidative burst at this location (Figure 7)

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