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NOD mice treated with MOTS-c showed a decreased abundance of Th1 cells and an increase in the abundance of Treg cells in the spleen and pancreas, which significantly delayed disease progression [8]

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Bile acids also promote colonic water secretion, through TGR5-mediated activation of adenylate cyclase, which stimulates chloride secretion through the cystic fibrosis transmembrane conductance regulator (CFTR) (174)

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Furthermore, copper deficiency impairs the activity of certain Cu/Zn-oxides, which may lead to increased accumulation of ROS and oxidative stress, which further promotes inflammation and atherosclerosis (104)

Bruce HR Wolffenbuttel About the author Bruce Wolffenbuttel is a Professor Emeritus MD PhD and an expert in endocrinology, metabolic diseases, and diabetes, with numerous published papers to his name