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doi: 10.3390/nu13061913
H-MESO-1 cells treated with TS (structure shown in Supplementary Fig
As the impaired mucosal antioxidative capacity may further promote oxidative damage, GSH deficiency might be a target for therapeutic intervention in IBD
Their electrophilic sulfur groups directly modify Keap1, activating the Nrf2 cytoprotective pathway, while concurrently suppressing NF-B-mediated inflammatory cascadesan effect not consistently observed in phenolic or alkaloid scaffolds (Egbujor et al., 2022
doi: 10.1038/s41419-018-1035-6 83 RambautAPybusOGNelsonMIViboudCTaubenbergerJKHolmesEC
