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In several prostate cancer cell lines, acetyl-l-carnitine acted as an anti-prostate cancer agent by inhibiting the production of chemokines CXCL12 and CCL2 as well as CXCR4 (chemokine ligand-receptor) and pro-inflammatory cytokines (IFN- and TNF-) [61]

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these include transient nausea and vomiting, abdominal cramps, and diarrhea

hypothesis: may be no difference between the two populations

highlight the importance of CrAT to contribute to maintaining a high metabolic plasticity of prostate cancers 22