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MT1 and MT2, which are rich in cysteine, can prevent copper toxicity by irreversible chelation within metal-sulfur salt clusters and can be induced by excess copper or other metals transcriptionally (56)

However, their application is limited by nonspecific toxicity

PKM2 and cancer: The function of PKM2 beyond glycolysis

It critically examines the direct correlations between these mechanisms, uses illustrative case studies to explore this duality in practice, and analyses the pivotal role of the aquatic environment in modulating both sides of the benefitrisk equation, a concept visually synthesised in Fig

Abstract Controlling tumor-specific alterations in metabolic pathways is a useful strategy for treating tumors

Evidence for peroxisomal dysfunction and dysregulation of the CDP-choline pathway in myalgic encephalomyelitis/chronic fatigue syndrome