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Rezin, G

Ginsenosides: The Need to Move Forward from Bench to Clinical Trials

Indeed, measurements of noradrenaline (NA)-induced aerobic glycolysis by FRET nanosensors in individual astrocytes demonstrated an increased glucose utilisation, manifested as reduced NA-elevated free cytosolic glucose concentration, whereas the production of lactate by NA was unaltered

Furthermore, SAE remains notably underdiagnosed and undertreated, and survivors often develop persistent long-term neurocognitive sequelae, including hippocampus-dependent memory impairment, psychiatric disorders and functional disability, which severely impact quality of life ( Oxidative stress is central to SAE pathogenesis, driven by an imbalance between excessive reactive oxygen species (ROS) production and impaired antioxidant defense systems (Yan et al., 2022)

Redox metabolism abnormalities in autistic children associated with mitochondrial disease

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