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Most of the GCL holoenzyme can be reversibly dissociated by treatment with dithiothreitol [42], while oxidative stress may enhance holoenzyme formation as it increases GCL activity in the absence of any change in the expression of GCL subunits [45]

Heterozygous parents and siblings of these patients are healthy with no symptoms, which clearly indicates an autosomal recessive disorder

Full text: Choi, I

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We will discuss several interesting aspects of the two figures

1994;266:E540547