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This interplay suggests that mitochondria-centered metabolic reprogramming may contribute to cellular senescence not only through alterations in metabolic pathways or metabolites but also by influencing cellular signaling and gene expression through mitochondrial enzymes and metabolites

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K-12045-D50, Advansta) for 2 min before being developed on a C-Digit (LI-COR Biosciences)

Additionally, a series of fluoroethyl Phe analogs have recently been developed as LAT1-targeted PET tracers

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