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Thrombotic arterial occlusion in the aftermath of plaque rupture is a critical cause of mortality, therefore the fact that oxLDL increases platelet aggregation (332-335) suggests an additional mechanism whereby elevated circulating oxLDL may increase risk of mortality during acute coronary events (336)

(a) Subcellular localization of HA-tagged ATGL S47A relative to ABHD5-mCitrine (mC) and lipid droplets

Grein, F

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