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MTHFR is a flavoprotein that facilitates NADPH-dependent reduction, utilizing FAD as a cofactor [1]

Sun Z, Tang Z, Yang X, Liu QS, Liang Y, Fiedler H, et al

In chronic respiratory settings, ACE2 downregulation, however, might stop SARSCoV-2 host cell contact (Restini et al., 2021)

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Its small size and endogenous origin make it a common target in mechanistic peptide research

Yet, riboflavin supplementation is increasingly explored in specific clinical settings, for example in case of preeclampsia 24 , anemia during pregnancy 25 , Parkinsons disease and migraine 26 , and female 27 , and postpartum depression 28 , as further discussed below