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Although mechanistic details are lacking, two scenarios to explain these observations are favored: Metabolically active intracellular parasites consume oxygen and decrease intracellular pH, both of which promote sickling of AS red cells

Evidently, intracellular cyst(e)ine contribute to ferroptosis suppression via at least two additional pathways in different contexts: in a mouse lung adenocarcinoma model, NFS1 cysteine desulfurase utilizes cysteine to synthesize ironsulfur clusters, which are necessary for preventing activation of the iron-starvation response, subsequent iron influx and ferroptosis 14 (Fig

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This improvement stems from the compounds ability to promote skin regeneration while reducing hyperpigmentation and sun damage