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D51.9 serves as the unspecified variant when clinical documentation fails to identify the deficiency mechanism

Data processing and presentation were conducted using BioRadCFXManager 3.0 (Bio-Rad, Hercules, CA) and flow cytometry software from ACEA Biosciences, United States

The different roles of glucocorticoids in the hippocampus and hypothalamus in chronic stress-induced HPA axis hyperactivity

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In the structure, different residues in the C-terminal pseudokinase domain of MLKL (S345/S347/T349 in mouse and S357/T358 in human) are phosphorylated by RIPK3, which induces a conformational change and promote the binding of inositol hexaphosphate (IP6) with positively charged patches in the N-terminal domain of MLKL [105,106,107]

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