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Indeed, to achieve adequate organ perfusion and offer much-needed hemodynamic support in the setting of contractile impairment during cardiac injury, overstimulation of the sympathetic nervous system initially develops as an adaptive neurohormonal response

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when Rev is provided in trans, cytoplasmic accumulation and post-transcriptional handling of HIV-1 RNA are sensed as a danger signal, activating innate immunity [15, 41, 42]

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However, robust modeling of hostmicrobiomepathogen interactions and materialtissue dynamics remains a major gap that must be filled through interdisciplinary research