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In parallel, inflammasome-related inflammatory pathways appear to be engaged after stroke in both the gut and the brain, and experimental post-stroke models link dysbiosis-driven barrier disruption and systemic inflammation to microglial NLRP3 inflammasome activation (

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Based on one study, which used immunohistochemistry to demonstrate the increased expression of HPGDS in the necrotic muscle fibers of patients with DMD and polymyositis but not in those of the small number of patients with FCMD 11 , HPGDS inhibitors have not been considered as therapeutic options in patients with FCMD

Consequently, histone modifications regulate gene expression by altering the chromatin structure and proteinprotein interaction

Naseer, Z., Ahmad, E., Aksoy, M

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