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These symptoms naturally result from the unique metabolic pathway of medium-chain fatty acids: rapid absorption and direct transport to the liver via the portal vein, where they undergo beta-oxidation and conversion into ketone bodies [1, 2]

The phosphatidylinositol 3-kinase/protein kinase B/mammalian target of rapamycin (PI3K/AKT/mTOR) signaling is a major pathway that activates glycolytic metabolism in B-cell non-Hodgkin lymphoma (B-NHL)

Lavallee, DC

One potential reason could the inability to increase muscle carnitine concentrations, although, in the case of Broad et al

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