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Notably, reverse experiments confirmed that downregulating SLC7A11 significantly increased baseline ROS levels and induced ferroptosis, offering a theoretical basis for targeting the SLC7A11 - GSH axis to reverse TMZ resistance
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This possibility should be considered if a sarcoidosis patient develops clinical-pathologic symptoms similar to those of sarcoidosis during treatment with a particular drug (i.e
J Neurochem 35: 12421245
Molecules, 19(11), 1906619077
K.TillyJ
