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This impaired gut barrier function and microbial imbalance can promote the translocation of bacteria, as well as pro-inflammatory cytokines and endotoxins (e.g., LPS), into systemic circulation [141, 142]

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Whereas preclinical and clinical research have extensively expounded on these target organ interventions, clinical application and instance-based evidence on clinical translatability are needed

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doi:10.1093/femsyr/fox028 ,

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