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There was full occlusion/occlusion-like syndrome, additionally presented with intracranial (superior sagittal sinus) hypertension, portal, and caval hypertension, aortal hypotension, progressed venous and arterial thrombosis peripherally and centrally, congested (i.e., inferior caval vein and superior mesenteric vein) and/or failed (i.e., azygos vein) major veins, and ECG disturbances

Conversely, patients with no visible pathology can have severe tendon pain

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These models provide a valuable ex vivo platform for evaluating pharmacological intervention, cell-based therapies and genetic manipulations aimed at enhancing remyelination for the treatment of IDDs