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The subsequent antigen presentation triggers the activation of Th1 and Th17 cells, leading to the release of pro-inflammatory cytokines (IL-2, TNF-, IFN-, IL-17), which in turn amplify oxidative stress and perpetuate an amplifying loop of inflammation and tissue damage (Kubiak et al

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TFM735 This compound was identified by high-throughput screening analysis as an activator of NRF2 through a C151-dependent mechanism [296]

a targeting moiety binds to EphA2 or EphA4 on cancer cells (e.g., EphA2 or EphA4 not bound to a ligand) rather than EphA2 or EphA4 on non-cancer cells (e.g., EphA2 or EphA4 bound to a ligand)

Our finding that CMS4 is overrepresented in PM sheds new light on the recently published results of the PRODIGE7 study, demonstrating a lack of benefit of intraperitoneal oxaliplatin treatment following cytoreductive surgery for PM [10]

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