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Other options in the GLP-1 space include combination approaches

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Of significance to diabetes is that ER stress-induced inflammation activates the kinases JNK (JUN N-terminal kinase) and IKK (inhibitor of nuclear factor-kappa B kinase subunit beta) that phosphorylate insulin receptor substrates (e.g

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387,388 These epigenetic changes contribute to the activation of pathogenic pathways involved in DPN