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Cellular glutathione (GSH) and glutathione s-transferase (GST) activity in human ovarian tumor biopsies following exposure to alkylating agents

Perilipin-null mice also display a significant resistance to diet-induced obesity (78, 106, 127) that is explained, at least in part, by increased beta-oxidation of fatty acids, the products of lipolysis (106), following from a compensatory induction of genes involved in lipid and energy metabolism and a downregulation of genes involved in lipid biosynthesis (20)

Bateman et al, 2013), but these acetylation events were not detected by our approach

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S Farhang Dehghan reports support for the present manuscript from Leona M

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