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Hiddemann W, Buechner T, Woermann B, Andreeff M

A., Dwyer, D

302 Elevated hepatic iron levels may lead to MAFLD and promote the progression of the disease, with excessive iron content may increase hepatocyte swelling, inflammation, and fibrosis, potentially converting isolated steatosis to MASH

This action might be linked to the upregulation of antioxidant enzymes, although this remains uncertain

Furthermore, we found that succinicGSH is secreted by FH-deficient cells and can be degraded, in a process similar to GSH catabolism, into succinic cysgly

and studies investigating the role of glutamatergic agonists to combat NMDAR hypofunction and reduce symptoms [7]