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Reprogramming in vivo produces teratomas and iPS cells with totipotency features
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A deficiency that causes sciatic nerve dysfunction generally disrupts nerve regeneration , slows nerve conduction , and inflames nerve pathways
Using a similar experimental approach, two laboratories showed that rMOR and hMOR were phosphorylated in the absence of agonist at S363 and T370 (Mouldous et al., 2012
10.4103/1673-5374.255974 261 LeeJ.ParkS
doi:10.1002/gps.4393 Tannenbaum C
