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doi: 10.1016/j.freeradbiomed.2007.03.033 85 ScharpfM.SchweizerU.ArzbergerT.RoggendorfW.SchomburgL.KohrleJ
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The most frequently cited mechanism is upregulation of vascular endothelial growth factor signaling (VEGFR2) together with modulation of the nitric-oxide system
The relationships between glutathione, glutathione-S-transferase and cytotoxicity of platinum drugs and melphalan in eight human ovarian carcinoma cell lines
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proposed two mechanisms by which (i) melatonin pretreatment represents an interfering mechanism for Ripk3/PGAM5/CypD axis execution, desensitizing cells to necroptosis triggered by RipK3 activation, PGAM upregulation, and CypD phosphorylation 50 in endothelial cells
