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Mechanistically, lanosterol administration increased the solubility of lens proteins and reduced oxidative stress by enhancing total antioxidant capacity and decreasing GSSG/GSH ratio in the lens cortex (150), and its effect was dependent on the severity of the condition or the lanosterol concentration in aqueous humor which varies from 0 to 31.61 ng/ml (150,153)

[10] (Table 4) was conducted without premedication and proceeded without any adverse effects

Studies of the biochemical pharmacology of the fermentation-derived antitumor agent, (Alpha s, 5s)-Alpha-Amino-3-Chloro-4,5-Dihydro-5-Isoxazoleacetic acid (at-125)

Hypertension and atrial fibrillation: doubts and certainties from basic and clinical studies

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