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It is different from TB-500 Research Peptide, different from GHRH analogues and different from metabolic receptor agonists

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Danial NN, Gramm CF, Scorrano L, Zhang C-Y, Krauss S, Ranger AM, et al

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References Costa, T

Dysregulation of KEAP1-NRF2 activity has been extensively demonstrated in cellular and animal models of ALS and in postmortem ALS motor cortex and spinal cord samples, so dysregulation of the NRF2/ARE antioxidant and cytoprotective pathway could be a possible mechanism underlying the progressive neurodegeneration in ALS and severity of the disease