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Because a reduction of glutathione levels in cells has been found to increase the risks for diseases and poisoning, direct intravenous injection of glutathione has been used to treat patients with chronic liver diseases and poisoning [8, 9]

Overall, this results in increased oxidative stress and decreased glutathione-based antioxidant capacity in these mice [40]

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Notably, in chronic heart failure, chemoreceptor reflex sensitivity abnormally increases, leading to excessive sympathetic nerve activation and accelerating myocardial remodeling (Sun et al., 1999)