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doi: 10.1002/mnfr.202100014

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Breaking this cycle requires potent, targeted interventions that can neutralize free radicals while simultaneously repairing the underlying mitochondrial machinerya dual action that makes compounds like ALCAR so clinically relevant

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doi: 10.1159/000341934

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Inhibition of miR-29b suppresses MAPK signaling pathway through targeting SPRY1 in atherosclerosis

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The mechanisms and potential therapeutic applications described remain under investigation and are not established for routine clinical use

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In addition, cancer cells chronically exposed to an acidic pH reveal a downregulation of the mitochondrial ACC2 isoform, the enzyme that regulates CPT1 activity by malonyl-CoA synthesis

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