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doi: 10.1002/mnfr.202100014

Breaking this cycle requires potent, targeted interventions that can neutralize free radicals while simultaneously repairing the underlying mitochondrial machinerya dual action that makes compounds like ALCAR so clinically relevant

doi: 10.1159/000341934
Inhibition of miR-29b suppresses MAPK signaling pathway through targeting SPRY1 in atherosclerosis

The mechanisms and potential therapeutic applications described remain under investigation and are not established for routine clinical use

In addition, cancer cells chronically exposed to an acidic pH reveal a downregulation of the mitochondrial ACC2 isoform, the enzyme that regulates CPT1 activity by malonyl-CoA synthesis
