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Consequently, it can be hypothesised that the administration of NAD + -boosting drugs may result in the regulation of ChREBP by means of pathways other than those involving the NAD +/Sirtuin pathways
This mechanism not only ensures targeted drug delivery but also allows for diffusion to adjacent non-targeted cells, resulting in potential bystander effects that enhance therapeutic efficacy [3]
BI 456906: discovery and preclinical pharmacology of a novel GCGR/GLP-1R dual agonist with robust anti-obesity efficacy
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Senescent cells rely on autophagy for maintenance and survival
