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In vivo deletion of -cell drp1 impairs insulin secretion without affecting islet oxygen consumption
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FIGURE 2 This oxidative stress environment can activate immune signalling pathways promoting neuroinflammation through mitochondrial dysfunction and impaired mitophagy, as observed in recent studies on ASD brains (Pinto Payares et al., 2024)
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Manipulations of MeCP2 in glutamatergic neurons highlight their contributions to Rett and other neurological disorders
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