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indicates that PAC, in the absence of adequate, physiological levels of GSH, may give rise to genotoxic quinone imine metabolites ( The production of quinone imine metabolite is the primary responsible for PAC liver and kidney toxicity

When introduced into the respiratory system, this sulfhydryl group directly interacts with the mucin polymers and performs a targeted disulfide exchange reaction

Another highly central theme, H, spans multiple domains, bridging kidney disease, chronic intermittent hypoxia, and antioxidant mechanisms

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The reverse bias has been reported to cause overheating

[DOI] [PubMed] [Google Scholar] 226.Sies H, Stahl W