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Description
Recent advances in our understanding of the pathogenesis of these tumors have indicated that defects in merlin are responsible for both sporadic and genetically acquired schwannomas, and the mechanisms by which merlin loss triggers tumor development are being unraveled (see summary in Figure 7)
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doi: 10.1016/0190-9622(95)91329-7
This targets resistant lateral fat deposits that affect body proportions
PMID: 28281554
Palavicini et al
