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Tsimikas S, Witztum JL, Miller ER, Sasiela WJ, Szarek M, Olsson AG, Schwartz GG

Moreover, a combination of structure-guided inhibitor and mutational analyses demonstrated that elongation activity, exemplified by TTLL6, is encoded by distinct active-site features that favor -linked glutamate chain propagation [29]

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Therefore, mitochondrial iron accumulation in AT2 cells may represent a crucial factor driving epithelial damage and pulmonary fibrosis

doi: 10.1089/ars.2005.7.508