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Inflammation, oxidative stress, Ca 2+ dysregulation, and hypoxia can induce cellular endoplasmic reticulum stress (ERS) by overloading the ERs protein folding mechanism ( Some studies have found that ferroptosis and ERS share a common pathway, especially in tumor diseases, and disease trends can be altered by controlling the ERS or ferroptosis pathway ( 2 Ferroptosis in RA With the study of RA disease mechanisms, it has been found that the aberrant activation of RA-FLSs, destruction of articular cartilage, overactivation of immune cells, and sustained expression of inflammatory factors are associated with ferroptosis

First is a reactive oxygen species (ROS)-centered model of intestinal immune regulation

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Acknowledgements Work discussed in this Review was supported by a Wellcome Trust Senior Investigator Award (095531), the Knut and Alice Wallenberg Foundation, the Swedish Research Council, the Hartwell Foundation for Biomedical Research (201500731), the Juvenile Diabetes Research Foundation (CDA-2-2013-54) and the US NIH (DK110276)