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10.1002/advs.202303259 33

10.1016/j.jaci.2016.07.029 J

The vitamin is then transferred to TC2, which serves as the circulating plasma transporter, hereditary defects in the production of TC2 and its receptor may produce functional deficiencies in B 12 and infantile megaloblastic anemia, and abnormal B 12 related biochemistry, even in some cases with normal blood B 12 levels

Paganoni S, Macklin EA, Hendrix S, Berry JD, Elliott MA, Maiser S, et al
