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Among these, syn oligomers (synOs) are considered highly neurotoxic due to their ability to induce mitochondrial dysfunction, impair synaptic transmission, damage cellular membranes, and trigger intracellular inflammation and lysosomal stress [11]

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Yegorov et al., 2020)

For glutathione, some people prefer "liposomal" forms or precursors like N-acetyl cysteine (NAC), which the body uses to build its own glutathione

This oxidant stress causes activation of a MAP kinase cascade in the cytosol ultimately leading to phosphorylation of c-jun N-terminal kinase (P-JNK), translocation of P-JNK to the mitochondria [34], and binding to the anchor protein Sab [35] for amplification of the mitochondrial oxidant stress and peroxynitrite formation in the mitochondrial matrix [33, 36]

NAC administration restored these changes in aspartame-treated mice leading to normal levels of all metabolites of the trans-sulphuration pathways