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Down-stream, dendritic cells exposed to smoke migrate poorly to lymph nodes and bias T-cell priming away from protective Th1 immunity, favouring Th2, Th17, and regulatory T-cell profiles marked by exhaustion receptors such as PD-1 and CTLA-4 (McNab et al., 2011
doi: 10.1089/ars.2010.3275
doi: 10.1093/humupd/dml054
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