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Researchers investigate GHK-Cu for its involvement in copper-associated signaling pathways, gene-expression regulation, extracellular matrix biology, and cellular communication processes

By concurrently depleting glutathione and perturbing copper homeostasis, EsCu@NPs elicit reinforced cuproptosis stress that is converted into immunogenic cell death

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Here, Cui and colleagues discuss the mechanisms that lead to leptin and ghrelin resistance, and how they might be exploited as targets for the management and treatment of obesity.