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Description
H., Cho K
Pharmacological targeting of CHAC1-mediated processes therefore represents a rational therapeutic strategy, potentially disrupting the cycle of GSH depletion, ER stress amplification and iron-dependent peroxidation that drives neuronal loss
The diagnostic paradigm of DN centers on persistent albuminuria and progressive glomerular filtration rate deterioration, establishing albuminuria reduction as a critical treatment endpoint (8, 9)
Garcia-Borja, E
These findings suggest that it is not necessarily the presence of the parasite cysts in the brain, but the downstream hormonal alterations, particularly in gonadal steroids, that mediate behavioral change
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