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Description
XBP1-deficient T cells in mice enhance antitumor immunity, suggesting their therapeutic potential in targeting this pathway

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Redox enzymes, such as Tv TrxR, are essential for maintaining oxidative balance, yet their integration within broader antioxidant networks may allow partial functional compensation under selective pressure [20, 133]

Metal organic framework-based antibacterial agents and their underlying mechanisms

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If validated, it would represent a new class of gene regulators distinct from transcription factors, microRNAs, and epigenetic modifiers